Jackson, B.J. and Eisenmann, D. M. 2012. Beta-catenin dependent Wnt signaling in C. elegans: Teaching an old dog a new trick. Cold Spring Harbor Perspectives in Biology. doi: 10.1101/cshperspect.a007948.
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D. M. Eisenmann. (2011) C. elegans seam cells as stem cells: Wnt signaling and casein kinase Iα regulate asymmetric cell divisions in an epidermal progenitor cell type. Cell Cycle 10: pg 21. Comment on: Banerjee D, et al. Cell Cycle 2010; 9:4748-65)

J. E. Gleason, and D. M. Eisenmann (2010) Wnt signaling controls the stem cell-like asymmetric division of the epithelial seam cells during C. elegans larval development. Developmental Biology 348, pages 58-66.

Gleason, J. E., E. A. Szyleyko and D. M. Eisenmann (2006) Multiple redundant Wnt signaling components function in two processes during C. elegans vulval development. Developmental Biology 298:442-457.
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J. B. Weidhaas, D. M. Eisenmann, J.M. Holub and S. Nallur (2006). A C. elegans tissue model of radiation-induced reproductive cell death. Proceedings National Academy of Sciences 103: 9946-9951.
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J. B. Weidhaas, D. M. Eisenmann, J.M. Holub and S. Nallur (2006). A Conserved EGFR/Ras/MAPK Pathway Regulates DNA Damage-Induced Cell Death Post-irradiation in Radelegans. Cancer Research 66: 10434-10438.
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Wagmaister, J.A., Miley, G.R., Morris, C.A., Gleason, J. E., Miller, L.M., Kornfeld, K., and D. M. Eisenmann. (2006). Identification of cis-regulatory elements from the C. elegans Hox gene lin-39 required for embryonic expression and for regulation by the transcription factors LIN-1, LIN-31 and LIN-39. Developmental Biology 297: 550-565.
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Wagmaister, J.A., Gleason, J. E., and D. M. Eisenmann. 2006. Transcriptional upregulation of the C. elegans Hox gene lin-39 during vulval cell fate specification. Mechanism of Development 123: 135-150.
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Eisenmann, D. M. 2005. Signal Transduction: Wnt Signaling. Chapter in WormBook, ed. The C. elegans Research Community, WormBook, doi/10.1895/wormbook.1.7.1, http://www.wormbook.org.
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Natarajan, L., B. M. Jackson, E. Szyleyko** and D. M. Eisenmann. 2004. Identification of evolutionarily conserved promoter elements and amino acids required for function of the C. elegans beta-catenin homolog BAR-1. Developmental Biology 272: 536-557.
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Joshi, P. and D. M. Eisenmann. 2004. The C. elegans pvl-5 gene protects hypodermal cells from ced-3-dependent, ced-4-independent cell death. Genetics 167: 673-685.
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Koh, K., Peyrot, S. M.**, Wood, C. G., Wagmaister, J. A., Eisenmann, D. M., and J. H. Rothman. 2002. Cell fates and fusion in the C. elegans vulval primordium are regulated by the EGL-18 and ELT-6 GATA factors – apparent direct targets of the LIN-39 Hox protein. Development 129: 5171-5180.
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Gleason, J, H. C. Korswagen, and D. M. Eisenmann. 2002. Activation of Wnt signaling can bypass the requirement for Ras signaling during C. elegans vulval induction. Genes and Development 16: 1281-1290.
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Natarajan, L., N. Witwer**, and D. M. Eisenmann. 2001. The divergent C. elegans beta-catenin proteins BAR-1, WRM-1 and HMP-2 make distinct protein interactions but maintain functional redundancy in vivo. Genetics 159: 159 –172.
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Eisenmann, D.M. and S.K. Kim. 2000. Protruding vulva mutants identify novel loci and Wnt signaling factors that function during C. elegans vulval development. Genetics 156: 1097-1116.
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Eisenmann, D.M. , J.N. Maloof, J. S. Simske, C. Kenyon and S.K. Kim. 1998. The ?-catenin homolog BAR-1 and LET-60 Ras coordinately regulate the Hox gene lin-39 during Caenorhabditis elegans vulval development. Development 125: 3667-3680.
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Eisenmann, D.M. and S.K. Kim. 1997. Mechanism of Activation of the C. elegans ras homologue let-60 by a novel, temperature-sensitive, gain-of-function mutation. Genetics 146: 553-565.
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Stolinski, L.A., Eisenmann, D.M., and Arndt, K.M. 1997. Identification of RTF1, a novel gene important for TATA site selection by TATA box-binding protein in Saccharomyces cerevisiae. Molecular and Cellular Biology 17: 4490-4500
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Eisenmann, D.M., C. Chapon, S.M. Roberts, C. Dollard, and F. Winston. 1994. The Saccharomyces cerevisiae SPT8 gene encodes a very acidic protein that is functionally related to SPT3 and TATA-binding protein. Genetics 137: 647-657.
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Eisenmann, D.M., K.A. Arndt, S.L. Ricupero, J.W. Rooney, and F. Winston. 1992. SPT3 interacts with TFIID to allow normal transcription in Saccharomyces cerevisiae. Genes and Development 6: 1319-1331.
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Arndt, K.M., S.L. Ricupero, D.M. Eisenmann, and F. Winston. 1992. Biochemical and genetic characterization of a yeast TFIID mutant that alters transcription in vivo and DNA-binding in vitro. Molecular and Cellular Biology 12: 2372-2382.
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Eisenmann, D.M., C. Dollard, and F. Winston. 1989. SPT15, the gene encoding the yeast TATA-binding factor TFIID, is required for normal transcription initiation in vivo. Cell 58: 1183-1191.
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Eisenmann, D.M. and S.K. Kim. 1994. Signal transduction and cell fate specification during Caenorhabditis elegans vulval development. Current Opinion in Genetics and Development 4: 508-516.
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